HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS		QUICK SEARCH:   [advanced] Author: Keyword(s): Year:  Vol:  Page:

This Article Full Text Full Text (PDF) Purchase Article View Shopping Cart Services Email this article to a friend Similar articles in this journal Alert me to new issues of the journal Download to citation manager Request Permissions Citing Articles Citing Articles via Google Scholar Google Scholar Articles by Herman, W. H. Articles by Engelgau, M. M. Search for Related Content PubMed Articles by Herman, W. H. Articles by Engelgau, M. M. Social Bookmarking                What's this?

Postprandial Hyperglycemia: Actor or Understudy?

With the recent development of new methods to measure postprandial hyperglycemia and new treatments to modulate it, investigators have questioned whether postprandial hyperglycemia causes diabetic complications and whether it should be a target of therapy. To address these questions, the American Diabetes Association convened a consensus development conference in January 2000. The conference report is reprinted on the following pages.1

In nondiabetic individuals, plasma glucose concentrations are extremely tightly regulated. Fasting and preprandial glucose concentrations range from 70 to 90 mg/dl. Glucose concentrations peak 60 min after the start of a meal, rarely exceed 120–140 mg/dl, and return to preprandial levels within 2–3 h. In diabetes, glucose concentrations vary widely both during a 24-h period and from day to day. For this reason, HbA_1c, which provides . . . [Full Text of this Article]

Footnotes

REFERENCES

CiteULike    Del.icio.us    Digg    Reddit    Technorati    What's this?